Why some people are gay: No gay gene? Make up an new cause and term “Epigenetics” and lead more people astray with pseudoscientific lies

Rather than accept the idea that homosexuality is an emotional illness, as it was viewed by psychiatry for most of human history or as a consequence of sin, as viewed by people of faith, scientists, liberals all; and having a leftist political agenda must, if necessary, manufacture out of nothing a “kind of” genetic fault and I direct your attention to the words they always use, like “they may” be caused by this epigenetic factor. (a) First, they cannot fund that gay gene; it does not seem to exist. (b) So, they invent a “kind of” gene, epi-marks or triggers that cause some heterosexuals to have gay desires.

Exodus 20:5 – “You shall not bow down to them or worship them; for I, the LORD your God, am a jealous God, punishing the children for the sin of the fathers to the third and fourth generation of those who hate me.” (NIV) Can the gross sexual sins of an unrepentant father cause severe emotional problems for their children and could this account for homosexuality? I don’t think there is any doubt that the guilt produced makes the man less of a husband and father, often leaving the children to be cared for by domineering mothers and could result in serious damage to the psyche of their children; and, it might be a link in the environmental causation of sexual problems with their children and that, often continues for several generations.

Yet, I understand how a mostly God rejecting world will refuse to accept the consequences of sin as an answer and explore the possibilities these sins can have on children for many generations. So, we are left to either push for gays to receive psychiatric help, leave the gays to self-loathing and worsening sexual practices and a life of sexual bondage; or, force some false scientific explanation, call homosexuality normal and pretend it is all okay. The last is the refuge of liberals, atheists and liberal science especially. This study proves nothing, it is all speculation.

New study claims homosexual trait is passed from mothers to sons and from fathers to daughters

• New study answers puzzle of why homosexuality seems to run in families
• It would not be expected to persist given Darwinian natural selection
• Researchers show it is an epigenetic rather than a genetic trait

From an evolutionary standpoint, homosexuality is a trait that would not be expected to develop and persist in the face of Darwinian natural selection.
It is nevertheless common for men and women in most cultures and previous studies have shown that homosexuality runs in families, leading most researchers to presume a genetic underpinning of sexual preference.

Puzzle: Scientists had long puzzled over why homosexuality appears to run in families when as a genetic trait it would not be expected to develop and persist in the face of Darwinian natural selection

However, no major gene for homosexuality has been found despite numerous studies searching for a genetic connection.

Now in a study published yesterday in the Quarterly Review of Biology, researchers from the U.S. National Institute of Mathematical and Biological Synthesis suggest homosexuality has an epigenetic link, not a genetic one.
Epigenetics explains how gene expression is regulated by temporary switches, called epi-marks. These constitute an extra layer of information attached to our genes’ backbones that determine their effect on our development.

While genes hold the instructions, epi-marks direct how those instructions are carried out – when, where and how much a gene is expressed during development.

William Rice, an an evolutionary biologist at the University of California Santa Barbara and lead author of the study, says epi-marks can determine the development of homosexuality in the offspring of heterosexual parents.
Sex-specific epi-marks produced in early foetal development protect each sex from the substantial natural variation in testosterone that occurs during later foetal development, the researchers say.

Sex-specific epi-marks stop girl foetuses from being masculinised when they experience atypically high testosterone, and vice versa for boy foetuses.
However, when these epi-marks are transmitted across generations from fathers to daughters or mothers to sons, they may cause reversed effects, such as the feminisation of some traits in sons, such as sexual preference, and similarly a partial masculinisation of daughters.

‘Transmission of sexually antagonistic epi-marks between generations is the most plausible evolutionary mechanism of the phenomenon of human homosexual.


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